Download e-book for iPad: Acute Renal Failure: Pathophysiology, Prevention, and by Vittorio E. Andreucci (auth.), Vittorio E. Andreucci (eds.)
By Vittorio E. Andreucci (auth.), Vittorio E. Andreucci (eds.)
`No doubt than it good merits its position within the library of the younger and not more younger nephrologists either regularly within the medical perform or in research.'
`The ebook is an up to date and helpful compendium of present wisdom within the medical administration of acute renal failure.'
Journal of the Royal Society of drugs, seventy eight (1985)
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Additional resources for Acute Renal Failure: Pathophysiology, Prevention, and Treatment
Recent studies, in fact, have demonstrated that thromboxane A2 (TxA 2) may play an important role in increasing the vascular resistance in this model of ARF [l83, l84}. Thus, the kidneys of rabbits treated with subcutaneous glycerol exhibited, 24 hours after the injection, an increased capacity to produce TxA2 [l83}' These findings have suggested a possible alteration in the relative proportion of vasodilating and vasoconstricting PGs in glycerolinduced ARF in rabbits with a predominance of vasoconstrictor substances; hence, the protective effect observed with i.
9% saline) has been shown not to protect rats against glycerolinduced ARF, although it decreased BUN by increasing urea clearance; inulin clearance was, in fact, severely reduced 24 and 48 hours after glycerol injection [155}. b. Immunization with antibodies against renin or All [168, l73} and the use of converting enzyme inhibitors or All blockers [175-l77} have failed to protect the rats against the renal damage by glycerol. c. If intrarenal RAS plays an important role in the development of glycerol-induced ARF, a kidney with high renin content is expected to be more susceptible to the development of ARF by glycerol.
Long-term (five to six weeks) sodium depletion induced a significant increase in renal cortical renin content in rats; yet the animals were protected against UN- or HgClrinduced ARF by short-term (48 hours) increase in NaC 1 intake and/or excretion [228}. In two-kidney Goldblatt rats, the ARF due to either arterial occlusion [127} or UN [127}, or HgCl 2 [232}, or glycerol [178} was equal in severity in both kidneys despite different degrees of renal cortical renin content. The increased solute excretion induced in rats by pretreatment with diuretics clearly protected the animals against HgClrinduced ARF, although no change in renal renin content had occurred [233}.
Acute Renal Failure: Pathophysiology, Prevention, and Treatment by Vittorio E. Andreucci (auth.), Vittorio E. Andreucci (eds.)